Intracellular IL-1 alpha in fibroblasts as a possible endogenous mediator of joint damage in rheumatoid arthritis

Isr J Med Sci. 1997 Apr;33(4):285-8.

Author

R N Apte¹

Affiliation

¹ Department of Microbiology and Immunology, Faculty of Health Sciences, Ben-Gurion University of the Negev, Beer Sheva, Israel.

PMID: 9347881

No abstract available

Publication types

Research Support, Non-U.S. Gov't
Review

MeSH terms

Animals
Arthritis, Rheumatoid / immunology*
Autoimmune Diseases / immunology*
Cell Division / immunology
Cellular Senescence / immunology
Fibroblasts / immunology*
Gene Expression Regulation
Humans
Inflammation Mediators / immunology*
Interleukin 1 Receptor Antagonist Protein
Interleukin-1 / genetics
Interleukin-1 / immunology*
Joints / immunology*
Receptors, Interleukin-1 / antagonists & inhibitors
Receptors, Interleukin-1 / immunology
Sialoglycoproteins / immunology

Substances

IL1RN protein, human
Inflammation Mediators
Interleukin 1 Receptor Antagonist Protein
Interleukin-1
Receptors, Interleukin-1
Sialoglycoproteins