Antineutrophil Cytoplasmic Autoantibodies and Associated Diseases: A Review
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Cited by (288)
Nonneoplastic Diseases of the Kidney
2020, Urologic Surgical PathologyANCA-Associated Vasculitis: Microscopic Polyangiitis, Eosinophilic Granulomatosis With Polyangiitis (Churg-Strauss Syndrome) and Granulomatosis With Polyangiitis (Wegener's Granulomatosis)
2017, The Heart in Rheumatic, Autoimmune and Inflammatory Diseases: Pathophysiology, Clinical Aspects and Therapeutic ApproachesDiagnostic Pathology: Head & Neck
2017, Diagnostic Pathology: Head and NeckAntineutrophil Cytoplasmic Antibody Associated Vasculitis
2015, Textbook of Pediatric RheumatologyPerformance of two strategies for urgent ANCA and anti-GBM analysis in vasculitis
2014, European Journal of Internal MedicineCitation Excerpt :Because of this possible rapid deterioration, patients who are suspected of AAV may benefit from rapid testing for ANCA and anti-glomerular basement membrane (GBM) antibodies to start immunosuppressive therapy as soon as a diagnosis is serologically supported and to differentiate these diseases from other conditions. The classical method to detect presence of ANCA is indirect immunofluorescence (IIF) on ethanol-fixed neutrophils which may show a C-ANCA (granular cytoplasmatic) pattern or P-ANCA (perinuclear staining) pattern [1–3]. In patients with AAV, these patterns are usually associated with antibodies against proteinase 3 (PR3) and myeloperoxidase (MPO) which are strongly associated with the presence of granulomatosis with poly-angiitis (GPA) or microscopic poly-angiitis (MPA) [3–5].
Autoimmune Disease in the Kidney
2013, The Autoimmune Diseases: Fifth Edition
Supported in part by grants from the National Institutes of Health (DK 40208) and the Jessie Ball DuPont Religious, Charitable, and Educational Foundation.