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The association between intestinal microbiome and autoimmune uveitis
2022, Archivos de la Sociedad Espanola de OftalmologiaMolecular determinants of peaceful coexistence versus invasiveness of non-Typhoidal Salmonella: Implications in long-term side-effects
2021, Molecular Aspects of MedicineCitation Excerpt :Notably, in the developing world, one third Reactive arthritis (ReA) and undifferentiated spondyloarthropathy (uSpA) patients are associated to Salmonella infection. Salmonella Lipopolysaccharide (LPS), antibodies against Salmonella and its DNA have been detected in the synovial cells and fluids of these patients indicating the involvement of Salmonella in causing these inflammatory conditions (Granfors et al., 1990). In the systemic investigation in 1995, out of 423 individuals infected with Salmonella food poisoning outbreak in 1984, 27 have detected the presence of Salmonella LPS using IgA anti-LPS antibody (Thomson et al., 1995).
Juvenile idiopathic arthritis and the gut microbiome: Where are we now?
2019, Best Practice and Research: Clinical RheumatologyCitation Excerpt :Translocation of bacterial antigen due to increased gut permeability can also lead to inflammation. In reactive arthritis following gastrointestinal infection, Yersinia antigens as well as Salmonella LPS could be detected in synovium suggesting the systemic dissemination of microbial antigens [58,59]. More details about the mechanism is available in the previous chapters.
Gut microbiota in children and altered profiles in juvenile idiopathic arthritis
2019, Journal of AutoimmunityCitation Excerpt :It was hypothesized that, during intestinal inflammation, increased mucosal uptake of gut bacteria and their membrane molecules, such as Lipopolysaccharides (LPS), peptidoglycan-polysaccharides (PG-PS) lead to systemic diffusion of these bacterial components and subsequent extra-intestinal inflammation, such as arthritis [125]. This hypothesis was also supported by historical studies showing reactivation of arthritis by systemically injected LPS or PG-PS [126,127] and the founding of bacterial antigens, such as that of Yersina spp. and Salmonella spp., within synovial leukocytes in patients with B27-associated reactive arthritis [128,129]. Some studies demonstrated that, not necessarily pathogenic bacteria, but also gut microbial communities are sufficient to induce joint inflammation [122,130].
Reactive arthritis in relation to internal derangements of the temporomandibular joint: a case control study
2015, British Journal of Oral and Maxillofacial Surgery