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We have read the editorial from Toes and Pisetsky1 and the letter from Holmdahl2 concerning the role of ACPA in the pathogenesis of RA.
With regards to the editorial, we appreciate the comments on how methodological hurdles may impact the interpretation of functional data. We like to add to this discussion that we have recently published data using new extensively characterised monoclonal ACPAs3 4 and new …
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