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Response to: ‘Inflammatory and non-inflammatory triggers of acute coronary syndromes’ by Eyuboglu
  1. Helga Westerlind1,
  2. Marie Holmqvist1,
  3. Lotta Ljung1,2,
  4. Thomas Frisell1,
  5. Johan Askling1,3
  1. 1Clinical Epidemiology Division, Department of Medicine, Solna, Karolinska Institutet, Stockholm, Sweden
  2. 2Department of Public Health and Clinical Medicine/Rheumatology, Umeå University, Umeå, Sweden
  3. 3Department of Rheumatology, Theme Infection and Inflammation, Karolinska University Hospital, Stockholm, Sweden
  1. Correspondence to Dr Helga Westerlind, Department of Medicine, Karolinska Institutet, S-171 77 Solna, Sweden; Helga.Westerlind{at}

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We thank Dr Eyuboglu for his comments1 on our article ‘Siblings of rheumatoid arthritis patients are at increased risk for acute coronary syndrome’.2

Familial risks tell us if risk factors for a disease are shared within families. By studying familial risks, we can also investigate shared susceptibility between diseases. In our present study, we have, by using the full siblings of patients with rheumatoid arthritis (RA), created a study population that are enriched for both the genetic and (familial) environmental risk factors involved in RA. If any disease has an increased frequency in our …

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