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Correspondence
Rheumatoid arthritis, γδ T cells and bisphosphonates
  1. Maurizio Rossini,
  2. Giovanni Adami,
  3. Ombretta Viapiana,
  4. Luca Idolazzi,
  5. Angelo Fassio,
  6. Alessandro Giollo,
  7. Cristian Caimmi,
  8. Giovanni Orsolini,
  9. Davide Gatti
  1. Rheumatology Unit, Department of Medicine, University of Verona, Verona, Italy
  1. Correspondence to Prof. Maurizio Rossini, Rheumatology Unit, Department of Medicine, University of Verona, P.le Scuro 10, Verona 37134, Italy; maurizio.rossini{at}univr.it

https://doi.org/10.1136/annrheumdis-2017-212510

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    We read with great interest the results of the study of Mo et al.1 The authors suggest that γδ T cells are involved in the pathogenesis of rheumatoid arthritis (RA). The study showed a significant reduction, in patients with RA, of peripheral total γδ T cells (particularly Vδ2 T cells, the major population of peripheral blood γδ T cells). The percentage of peripheral Vδ2 T cells of RA was negatively correlated with the levels of inflammatory markers, including C-reactive protein, erythrocyte sedimentation rate as well as the Disease Activity Score in 28 joints. The peripheral reduction of Vδ2 …

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    Footnotes

    • Contributors All authors have contributed to all phases of the production of the manuscript.

    • Competing interests None declared.

    • Provenance and peer review Not commissioned; internally peer reviewed.

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