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  • Loss of phosphatase and tensin homolog (PTEN) in myeloid cells controls inflammatory bone destruction by regulating the osteoclastogenic potential of myeloid cells

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Basic and translational research
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Loss of phosphatase and tensin homolog (PTEN) in myeloid cells controls inflammatory bone destruction by regulating the osteoclastogenic potential of myeloid cells
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Authors

  1. Correspondence to Dr Kurt Redlich, Division of Rheumatology, Internal Medicine III, Medical University of Vienna, Währinger Gürtel 18-20, Vienna 1090, Austria; kurt.redlich{at}meduniwien.ac.at Gernot Schabbauer, Center for Biomolecular Medicine and Pharmacology, Medical University Vienna. A-1090 Vienna, Austria; gernot.schabbauer@meduniwien.ac.at
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Citation

Blüml S, Friedrich M, Lohmeyer T, et al
Loss of phosphatase and tensin homolog (PTEN) in myeloid cells controls inflammatory bone destruction by regulating the osteoclastogenic potential of myeloid cells
Annals of the Rheumatic Diseases 2015;74:227-233.

Publication history

  • Received February 18, 2013
  • Revised August 21, 2013
  • Accepted September 8, 2013
  • First published September 27, 2013.
Online issue publication 
March 11, 2024

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