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Response to: ‘Exacerbation of immune thrombocytopenia triggered by COVID-19 in patients with systemic lupus erythematosus’ by Kondo et al
  1. Alexis Mathian,
  2. Zahir Amoura
  1. Sorbonne Université, Assistance Publique–Hôpitaux de Paris, Groupement Hospitalier Pitié–Salpêtrière, French National Referral Center for Systemic Lupus Erythematosus, Antiphospholipid Antibody Syndrome and Other Autoimmune Disorders, Service de Médecine Interne 2, Institut E3M, Inserm UMRS, Centre d’Immunologie et des Maladies Infectieuses (CIMI-Paris), Paris, France
  1. Correspondence to Dr Alexis Mathian, Internal Medicine, University Hospital Pitié Salpêtrière, Paris 75651, France; alexis.mathian{at}aphp.fr

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We thank Kondo et al 1 for their interest in our study reporting on the course of severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) disease (COVID-19) in a case series of patients with systemic lupus erythematosus (SLE) under long-term treatment with hydroxychloroquine.2 Currently, there are no data that identify SLE as a risk factor for COVID-19-related immunological complications. Kondo et al 1 report a patient with SLE experiencing an exacerbation of immune thrombocytopaenic purpura (ITP) likely triggered by COVID-19. This case raises several subjects for discussion. In the cohort of patients with SLE that we reported, there were no manifestations of lupus activity during the course of COVID-19, except for one patient who had a tenosynovitis at the onset of SARS-CoV-2 …

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Footnotes

  • Handling editor Josef S Smolen

  • Contributors AM and ZA wrote the manuscript.

  • Funding The authors have not declared a specific grant for this research from any funding agency in the public, commercial or not-for-profit sectors.

  • Competing interests None declared.

  • Patient and public involvement Patients and/or the public were not involved in the design, or conduct, or reporting, or dissemination plans of this research.

  • Provenance and peer review Commissioned; internally peer reviewed.

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