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Elevated EPSTI1 promote B cell hyperactivation through NF-κB signalling in patients with primary Sjögren’s syndrome

Authors

  • Jin-lei Sun Department of Rheumatology and Clinical Immunology, Peking Union Medical College Hospital, Chinese Academy of Medical Sciences & Peking Union Medical College, Beijing, China Key Laboratory of Rheumatology and Clinical Immunology, Ministry of Education, Beijing, China PubMed articlesGoogle scholar articles
  • Hao-ze Zhang Department of Rheumatology and Clinical Immunology, Peking Union Medical College Hospital, Chinese Academy of Medical Sciences & Peking Union Medical College, Beijing, China Key Laboratory of Rheumatology and Clinical Immunology, Ministry of Education, Beijing, China PubMed articlesGoogle scholar articles
  • Su-ying Liu Department of Rheumatology and Clinical Immunology, Peking Union Medical College Hospital, Chinese Academy of Medical Sciences & Peking Union Medical College, Beijing, China Key Laboratory of Rheumatology and Clinical Immunology, Ministry of Education, Beijing, China PubMed articlesGoogle scholar articles
  • Chao-feng Lian Department of Rheumatology and Clinical Immunology, Peking Union Medical College Hospital, Chinese Academy of Medical Sciences & Peking Union Medical College, Beijing, China Key Laboratory of Rheumatology and Clinical Immunology, Ministry of Education, Beijing, China PubMed articlesGoogle scholar articles
  • Zhi-lei Chen Department of Rheumatology and Clinical Immunology, Peking Union Medical College Hospital, Chinese Academy of Medical Sciences & Peking Union Medical College, Beijing, China Key Laboratory of Rheumatology and Clinical Immunology, Ministry of Education, Beijing, China PubMed articlesGoogle scholar articles
  • Ti-hong Shao Department of Rheumatology and Clinical Immunology, Peking Union Medical College Hospital, Chinese Academy of Medical Sciences & Peking Union Medical College, Beijing, China Key Laboratory of Rheumatology and Clinical Immunology, Ministry of Education, Beijing, China PubMed articlesGoogle scholar articles
  • Shuo Zhang Department of Rheumatology and Clinical Immunology, Peking Union Medical College Hospital, Chinese Academy of Medical Sciences & Peking Union Medical College, Beijing, China Key Laboratory of Rheumatology and Clinical Immunology, Ministry of Education, Beijing, China PubMed articlesGoogle scholar articles
  • Li-ling Zhao Department of Rheumatology and Clinical Immunology, Peking Union Medical College Hospital, Chinese Academy of Medical Sciences & Peking Union Medical College, Beijing, China Key Laboratory of Rheumatology and Clinical Immunology, Ministry of Education, Beijing, China PubMed articlesGoogle scholar articles
  • Cheng-mei He Department of Rheumatology and Clinical Immunology, Peking Union Medical College Hospital, Chinese Academy of Medical Sciences & Peking Union Medical College, Beijing, China Key Laboratory of Rheumatology and Clinical Immunology, Ministry of Education, Beijing, China PubMed articlesGoogle scholar articles
  • Mu Wang Department of Stomatology, Peking Union Medical College Hospital, Chinese Academy of Medical Science & Peking Union Medical College, Beijing, China PubMed articlesGoogle scholar articles
  • Wen Zhang Department of Rheumatology and Clinical Immunology, Peking Union Medical College Hospital, Chinese Academy of Medical Sciences & Peking Union Medical College, Beijing, China Key Laboratory of Rheumatology and Clinical Immunology, Ministry of Education, Beijing, China PubMed articlesGoogle scholar articles
  • Hua Chen Department of Rheumatology and Clinical Immunology, Peking Union Medical College Hospital, Chinese Academy of Medical Sciences & Peking Union Medical College, Beijing, China Key Laboratory of Rheumatology and Clinical Immunology, Ministry of Education, Beijing, China PubMed articlesGoogle scholar articles
  • Feng-chun Zhang Department of Rheumatology and Clinical Immunology, Peking Union Medical College Hospital, Chinese Academy of Medical Sciences & Peking Union Medical College, Beijing, China Key Laboratory of Rheumatology and Clinical Immunology, Ministry of Education, Beijing, China PubMed articlesGoogle scholar articles
  1. Correspondence to Hua Chen, Department of Rheumatology and Clinical Immunology, Peking Union Medical College Hospital, Chinese Academy of Medical Sciences & Peking Union Medical College, Beijing 100730, China; chenhua{at}pumch.cn; Feng-chun Zhang; zhangfccra{at}aliyun.com
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Citation

Sun J, Zhang H, Liu S, et al
Elevated EPSTI1 promote B cell hyperactivation through NF-κB signalling in patients with primary Sjögren’s syndrome

Publication history

  • Received October 4, 2019
  • Revised January 18, 2020
  • Accepted February 5, 2020
  • First published February 29, 2020.
Online issue publication 
May 13, 2021

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