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PDFBasic and translational research
Extended report
Hypermetabolic macrophages in rheumatoid arthritis and coronary artery disease due to glycogen synthase kinase 3b inactivation
- Correspondence to Dr Cornelia M Weyand, Division of Immunology and Rheumatology, Stanford University School of Medicine, Stanford, CA 94305, USA; cweyand{at}stanford.edu
Citation
Hypermetabolic macrophages in rheumatoid arthritis and coronary artery disease due to glycogen synthase kinase 3b inactivation
Publication history
- Received November 3, 2017
- Revised January 13, 2018
- Accepted January 16, 2018
- First published February 3, 2018.
Online issue publication
April 10, 2023
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- Previous version (12 June 2018).
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© Article author(s) (or their employer(s) unless otherwise stated in the text of the article) 2018. All rights reserved. No commercial use is permitted unless otherwise expressly granted.