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08.14 IL-23 receptor signalling is critical in normal bone remodelling and influences osteoclast activity in vitro and t cell-driven inflammatory bone damage in vivo

Authors

  • Wida Razawy 1Department of Rheumatology, Erasmus Medical Centre, Rotterdam, The Netherlands2Department of Immunology, Erasmus Medical Centre, Rotterdam, The Netherlands PubMed articlesGoogle scholar articles
  • Patrick S Asmawidjaja 1Department of Rheumatology, Erasmus Medical Centre, Rotterdam, The Netherlands2Department of Immunology, Erasmus Medical Centre, Rotterdam, The Netherlands PubMed articlesGoogle scholar articles
  • Adriana MC Mus 1Department of Rheumatology, Erasmus Medical Centre, Rotterdam, The Netherlands2Department of Immunology, Erasmus Medical Centre, Rotterdam, The Netherlands PubMed articlesGoogle scholar articles
  • Marijke Schreuders-Koedam 3Internal Medicine, Erasmus Medical Centre, Rotterdam, The Netherlands PubMed articlesGoogle scholar articles
  • Marlieke Molendijk 1Department of Rheumatology, Erasmus Medical Centre, Rotterdam, The Netherlands2Department of Immunology, Erasmus Medical Centre, Rotterdam, The Netherlands PubMed articlesGoogle scholar articles
  • Mohamed Oukka 4Center for Immunity and Immunotherapies, Seattle Children’s Research Institute, Seattle, USA PubMed articlesGoogle scholar articles
  • Vijay K Kuchroo 5Evergrande Centre for Immunologic Diseases and Ann Romney Centre for Neurologic diseases, Harvard Medical School and Brigham and Women’s Hospital, Boston, USA6The Broad Institute of MIT and Harvard, Cambridge, USA PubMed articlesGoogle scholar articles
  • Bram CJ van der Eerden 3Internal Medicine, Erasmus Medical Centre, Rotterdam, The Netherlands PubMed articlesGoogle scholar articles
  • Erik Lubberts 1Department of Rheumatology, Erasmus Medical Centre, Rotterdam, The Netherlands2Department of Immunology, Erasmus Medical Centre, Rotterdam, The Netherlands PubMed articlesGoogle scholar articles

Citation

Razawy W, Asmawidjaja PS, Mus AM, et al
08.14 IL-23 receptor signalling is critical in normal bone remodelling and influences osteoclast activity in vitro and t cell-driven inflammatory bone damage in vivo

Publication history

  • First published March 1, 2017.
Online issue publication 
January 09, 2022

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