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Disease activity and left ventricular systolic function in rheumatoid arthritis
  1. Alessandro Giollo1,
  2. Giovanni Cioffi2,
  3. Federica Ognibeni1,
  4. Andrea Dalbeni3,
  5. Davide Gatti1,
  6. Luca Idolazzi1,
  7. Maurizio Rossini1,
  8. Ombretta Viapiana1
  1. 1Rheumatology Unit, Department of Medicine, University of Verona, Verona, Italy
  2. 2Department of Cardiology, Villa Bianca Hospital, Trento, Italy
  3. 3General Medicine and Hypertension Unit, Department of Medicine, University of Verona, Verona, Italy
  1. Correspondence to Dr Alessandro Giollo, Rheumatology Unit, Department of Medicine, University of Verona, Policlinico G.B. Rossi, Piazzale Ludovico Antonio Scuro 10, Verona 37134, Italy; alessandrogiollo{at}

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We have read with interest the recent paper by Midtbø et al.1 The authors evaluated 119 patients with rheumatoid arthritis (RA), and found that patients with RA active disease (Simplified Disease Activity Index (SDAI) >3.3) compared with those in remission (SDAI<3.3) had lower left ventricular (LV) systolic myocardial function, assessed by stress-corrected mid-wall shortening (scMWS) and global longitudinal strain (GLS).

Data from Midtbø et al are in line with our previous work, in which we demonstrated that RA per se is a condition closely related to LV systolic dysfunction (LVSD) assessed by scMWS.2 Furthermore, similar to other pathophysiological models (systemic hypertension, diabetes mellitus and aortic stenosis), we showed that the LVSD in patients with RA is closely …

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