Article info

Download PDFPDF
AB0103 Tacrolimus Inhibit IL-1β and ER Stress-Induced, Rankl-Mediated Osteoclastogenesis by Inhibiting Perk, Ire1, Grp78, Eif2a, C-Fos and Nfatc1

Authors

  • W.-S. Lee 1Division of Rheumatology, Department of Internal Medicine, Chonbuk National University Medical School and Research Institute of Clinical Medicine of Chonbuk National University Hospital-Chonbuk National University, jeonju PubMed articlesGoogle scholar articles
  • E.-G. Lee 1Division of Rheumatology, Department of Internal Medicine, Chonbuk National University Medical School and Research Institute of Clinical Medicine of Chonbuk National University Hospital-Chonbuk National University, jeonju PubMed articlesGoogle scholar articles
  • M.-S. Sung 1Division of Rheumatology, Department of Internal Medicine, Chonbuk National University Medical School and Research Institute of Clinical Medicine of Chonbuk National University Hospital-Chonbuk National University, jeonju PubMed articlesGoogle scholar articles
  • C.-H. Lee 2Department of Internal Medicine, School of medicine, Wonkwang university, Iksan, Korea, Republic Of PubMed articlesGoogle scholar articles
  • M.-S. Lee 2Department of Internal Medicine, School of medicine, Wonkwang university, Iksan, Korea, Republic Of PubMed articlesGoogle scholar articles
  • W.-H. Yoo 1Division of Rheumatology, Department of Internal Medicine, Chonbuk National University Medical School and Research Institute of Clinical Medicine of Chonbuk National University Hospital-Chonbuk National University, jeonju PubMed articlesGoogle scholar articles

Citation

Lee W, Lee E, Sung M, et al
AB0103 Tacrolimus Inhibit IL-1β and ER Stress-Induced, Rankl-Mediated Osteoclastogenesis by Inhibiting Perk, Ire1, Grp78, Eif2a, C-Fos and Nfatc1

Publication history

  • First published June 10, 2014.
Online issue publication 
April 13, 2016

Request permissions

If you wish to reuse any or all of this article please use the link below which will take you to the Copyright Clearance Center’s RightsLink service. You will be able to get a quick price and instant permission to reuse the content in many different ways.