Response to: ‘The influence of inflammation in the development of subclinical atherosclerosis in psoriatic arthritis’ by González-Gay et al

Michael T Nurmohamed; Anna Jamnitski; Deborah Symmons; Mike Johannes Leonardus Peters; Naveed Sattar; Iain McInnes

doi:10.1136/annrheumdis-2014-205174

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Correspondence response

Response to: ‘The influence of inflammation in the development of subclinical atherosclerosis in psoriatic arthritis’ by González-Gay et al

Michael T Nurmohamed1,
Anna Jamnitski2,
Deborah Symmons3,
Mike Johannes Leonardus Peters4,
Naveed Sattar5,
Iain McInnes6

¹Department of Rheumatology and Internal Medicine, VUMC, Amsterdam, The Netherlands
²Jan van Breemen Research Institute/READE, Amsterdam, The Netherlands
³Manchester, UK
⁴Department of Rheumatology, VU University Medical Center, Amsterdam, The Netherlands
⁵BHF Glasgow Cardiovascular Research Centre, University of Glasgow, Glasgow, UK
⁶Institute of Infection, Immunity and Inflammation, University of Glasgow, Glasgow, UK

Correspondence to Dr Michael T Nurmohamed, Department of Rheumatology and Internal Medicine, VUMC, PO Box 7057, Room 4A32, Amsterdam 1007 MB, The Netherlands; m.nurmohamed{at}reade.nl

https://doi.org/10.1136/annrheumdis-2014-205174

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In their comments about our systematic review on cardiovascular (CV) comorbidities in patients with psoriatic arthritis,1 Gonzalez-Gay et al2 point out that in their studies, in contrast to our table 4, they did demonstrate a correlation between inflammatory parameters and flow-mediated dilatation …

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