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Basic and translational research
Extended report
HRES-1/Rab4-mediated depletion of Drp1 impairs mitochondrial homeostasis and represents a target for treatment in SLE

Authors

  • Tiffany N Caza Departments of Medicine, Microbiology, and Immunology, Biochemistry and Molecular Biology, Neuroscience and Physiology, and Pathology, SUNY Upstate Medical University, Syracuse, New York, USA PubMed articlesGoogle scholar articles
  • David R Fernandez Departments of Medicine, Microbiology, and Immunology, Biochemistry and Molecular Biology, Neuroscience and Physiology, and Pathology, SUNY Upstate Medical University, Syracuse, New York, USA PubMed articlesGoogle scholar articles
  • Gergely Talaber Departments of Medicine, Microbiology, and Immunology, Biochemistry and Molecular Biology, Neuroscience and Physiology, and Pathology, SUNY Upstate Medical University, Syracuse, New York, USA PubMed articlesGoogle scholar articles
  • Zachary Oaks Departments of Medicine, Microbiology, and Immunology, Biochemistry and Molecular Biology, Neuroscience and Physiology, and Pathology, SUNY Upstate Medical University, Syracuse, New York, USA PubMed articlesGoogle scholar articles
  • Mark Haas Department of Pathology, Cedars-Sinai Medical Center, Los Angeles, California, USA PubMed articlesGoogle scholar articles
  • Michael P Madaio Department of Medicine, Medical College of Georgia, Augusta, Georgia, USA PubMed articlesGoogle scholar articles
  • Zhi-wei Lai Departments of Medicine, Microbiology, and Immunology, Biochemistry and Molecular Biology, Neuroscience and Physiology, and Pathology, SUNY Upstate Medical University, Syracuse, New York, USA PubMed articlesGoogle scholar articles
  • Gabriella Miklossy Departments of Medicine, Microbiology, and Immunology, Biochemistry and Molecular Biology, Neuroscience and Physiology, and Pathology, SUNY Upstate Medical University, Syracuse, New York, USA PubMed articlesGoogle scholar articles
  • Ram R Singh Department of Medicine, UCLA, Los Angeles, California, USA PubMed articlesGoogle scholar articles
  • Dmitriy M Chudakov Shemiakin-Ovchinnikov Institute of Bioorganic Chemistry, RAS, Moscow, Russia PubMed articlesGoogle scholar articles
  • Walter Malorni Department of Experimental Medicine, University of Rome, Rome, Italy PubMed articlesGoogle scholar articles
  • Frank Middleton Departments of Medicine, Microbiology, and Immunology, Biochemistry and Molecular Biology, Neuroscience and Physiology, and Pathology, SUNY Upstate Medical University, Syracuse, New York, USA PubMed articlesGoogle scholar articles
  • Katalin Banki Departments of Medicine, Microbiology, and Immunology, Biochemistry and Molecular Biology, Neuroscience and Physiology, and Pathology, SUNY Upstate Medical University, Syracuse, New York, USA PubMed articlesGoogle scholar articles
  • Andras Perl Departments of Medicine, Microbiology, and Immunology, Biochemistry and Molecular Biology, Neuroscience and Physiology, and Pathology, SUNY Upstate Medical University, Syracuse, New York, USA PubMed articlesGoogle scholar articles
  1. Correspondence to Dr Andras Perl, Department of Medicine, State University of New York, 750 East Adams Street, Syracuse, New York 13210, USA; perla{at}upstate.edu
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Citation

Caza TN, Fernandez DR, Talaber G, et al
HRES-1/Rab4-mediated depletion of Drp1 impairs mitochondrial homeostasis and represents a target for treatment in SLE
Annals of the Rheumatic Diseases 2014;73:1888-1897.

Publication history

  • Received April 16, 2013
  • Revised June 13, 2013
  • Accepted July 9, 2013
  • First published July 29, 2013.
Online issue publication 
October 25, 2017

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Published by the BMJ Publishing Group Limited. For permission to use (where not already granted under a licence) please go to http://group.bmj.com/group/rights-licensing/permissions This is an Open Access article distributed in accordance with the Creative Commons Attribution Non Commercial (CC BY-NC 3.0) license, which permits others to distribute, remix, adapt, build upon this work non-commercially, and license their derivative works on different terms, provided the original work is properly cited and the use is non-commercial. See: http://creativecommons.org/licenses/by-nc/3.0/