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COMP: a candidate molecule in the pathogenesis of systemic sclerosis with a potential as a disease marker
  1. R Hesselstrand1,
  2. A Kassner2,
  3. D Heinegård2,
  4. T Saxne1
  1. 1
    Department of Clinical Sciences, Section for Rheumatology, Lund University, Sweden
  2. 2
    Department of Experimental Medical Sciences, Section for Connective Tissue Biology, Lund University, Sweden
  1. R Hesselstrand, Department of Rheumatology, Lund University Hospital, S-221 85 Lund, Sweden; roger.hesselstrand{at}skane.se

Footnotes

  • RH and AK contributed equally to the study.

  • Funding: This study was supported by grants from the Swedish Medical Research Council, the Medical Faculty of Lund University, the Swedish Rheumatism Association, King Gustaf V 80-year Fund, the Österlund Foundation, the Kock Foundation and the European Community’s FP6 funding (AUTOCURE project) and the Anna-Greta Crafoord stiftelse (AK).

  • Competing interests: DH and TS are cofounders and minor shareholders of AnaMar Medical, Lund, Sweden. This publication only reflects the author’s views. The European Community is not liable for any use that may be made of the information herein.

  • Ethics approval: The study was approved by the regional ethics board.

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Footnotes

  • RH and AK contributed equally to the study.

  • Funding: This study was supported by grants from the Swedish Medical Research Council, the Medical Faculty of Lund University, the Swedish Rheumatism Association, King Gustaf V 80-year Fund, the Österlund Foundation, the Kock Foundation and the European Community’s FP6 funding (AUTOCURE project) and the Anna-Greta Crafoord stiftelse (AK).

  • Competing interests: DH and TS are cofounders and minor shareholders of AnaMar Medical, Lund, Sweden. This publication only reflects the author’s views. The European Community is not liable for any use that may be made of the information herein.

  • Ethics approval: The study was approved by the regional ethics board.

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