Diverse vascular lesions occur in about one third of patients with Behçet’s disease (BD). Endothelial dysfunction is known to have an important role in the development of these lesions.¹ Acute systemic inflammation and chronic systemic vasculitis are associated with endothelial dysfunction.^2,3 Moreover, inflammation is an important risk factor for future cardiovascular events.⁴ These findings have led to the hypothesis that acute and chronic inflammatory processes associated with BD may cause endothelial dysfunction, which can then lead to a subsequent increase of arterial stiffness and vascular damage that are closely related to the clinical course of BD. Arterial stiffness is a reliable predictor of subsequent cardiovascular mortality and vascular damage. Pulse wave velocity (PWV) is an ideal indicator of arterial stiffness.^5,6 …