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Anti-TNF antibody treatment improves glucocorticoid induced insulin-like growth factor 1 (IGF1) resistance without influencing myoglobin and IGF1 binding proteins 1 and 3
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    Myoglobin is not a marker of accellerated muscle protein breakdown

    Dear Editor,

    RA is a systemic disease and Sarzi-Puttini and colleagues have to be praised for investigating the effects of anti-TNF therapy on the circulating IGF system rather than the usual measures of disease activity, joint damage and disability. However, their conclusions are based on false assumption that serum myoglobin is a marker of the muscle catabolism characteristic of rheumatoid cachexia.

    In m...

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    Conflict of Interest:
    None declared.