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Asthenoazoospermia in patients receiving anti-tumour necrosis factor α agents
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Tumour necrosis factor α (TNFα) is known to affect gonadal cell viability and function by several pathways: inhibiting germ cell apoptosis1; promoting Sertoli cell androgen receptor expression and activity2; and inhibiting Leydig testicular cell steroidogenesis at the transcriptional level.3 Recently, infliximab (that is, …