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Obstruction of the pulmonary artery by granulomatous vasculitis: a clinical, morphological, and immunological analysis
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  1. Correspondence to:
    Dr G Schett, Division of Rheumatology, Department of Internal Medicine III, University of Vienna, Währinger Gürtel 18–20, A-1090 Vienna, Austria;
    georg.schett{at}akh-wien.ac.at
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Citation

Schett G, Winkler S, Hollenstein U, et al
Obstruction of the pulmonary artery by granulomatous vasculitis: a clinical, morphological, and immunological analysis

Publication history

  • Accepted December 10, 2001
  • First published May 1, 2002.
Online issue publication 
May 01, 2002
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    Web-only Figures W1 and W2
     
     
     
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    Figure W1 Positron emission tomography (fluorodeoxyglucose-PET). PET scans were performed before the start of immunosuppressive treatment. Both axial (a) and coronal sections (b) show an accumulation of tracer in the aortic wall as well as in the pulmonary artery.
     
     
     
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    Figure W2 Production of tumour necrosis factor alpha (TNF-alpha). FACS analysis of permeabilised peripheral blood mononuclear cells shows the intracellular TNF-alpha production. In normal subjects the number of TNF-alpha producing T cells is low, both in the CD4+ and in the CD8+ T cell subsets, whereas it returns to normal after immunosuppressive treatment.

     

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