Prospective study of the early course of rheumatoid arthritis in young adults: Comparison of patients with and without rheumatoid factor positivity at entry and identification of variables correlating with outcome☆
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Cited by (124)
Clinical Features of Rheumatoid Arthritis
2012, Kelley's Textbook of Rheumatology: Volume 1-2, Ninth EditionDevelopment of a scoring system for assessment of outcome of early undifferentiated inflammatory synovitis
2008, Joint Bone SpineCitation Excerpt :From the clinical perspective, if effective therapy could be introduced prior to the development of presumed irreversible damage, outcomes could be improved. This was fuelled by the “window of opportunity” hypothesis for therapeutic intervention in RA [8]. The hypothesis was based on the existence of a time frame within which there is potential for a disproportionate response to therapy, resulting in long-term benefits, or more importantly, the chance of “cure”.
Development of a scoring system for assessment of outcome of early undifferentiated inflammatory synovitis
2008, Revue du Rhumatisme (Edition Francaise)Autoantibodies in rheumatoid arthritis: a review
2006, Biomedicine and PharmacotherapyCitation Excerpt :There is evidence that they could contribute to disease by the formation of immune complexes [15] and complement fixation [16], and also by functioning as highly efficient antigen-presenting cells [17]. The correlation of RF status and progression of joint damage in RA [18] support a role in pathogenesis although they are by no means an absolute requirement for severe disease and joint destruction. Autoantibodies to citrullinated antigens such as anti-perinuclear factor (APF) [19], anti-keratin antibody (AKA) [20], anti-Sa [21] and anti-CCP antibodies [22] are an overlapping group of antibodies with a remarkable specificity completely dependent on the citrullination of arginine residues.
Are early arthritis clinics necessary?
2005, Best Practice and Research: Clinical RheumatologyAutoimmunity - Rheumatoid Arthritis
2005, Measuring Immunity: Basic Biology and Clinical Assessment
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Supported in part by Grants AM-12049 and AM-05055 of the National Institute of Arthritis, Metabolism, and Digestive Diseases, Bethesda, Md.
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From the Section of Rheumatology, Department of Medicine, University of Tennessee Center for the Health Sciences, Memphis, Tenn.