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Correspondence response
Response to: ‘The influence of inflammation in the development of subclinical atherosclerosis in psoriatic arthritis’ by González-Gay et al
  1. Michael T Nurmohamed1,
  2. Anna Jamnitski2,
  3. Deborah Symmons3,
  4. Mike Johannes Leonardus Peters4,
  5. Naveed Sattar5,
  6. Iain McInnes6
  1. 1Department of Rheumatology and Internal Medicine, VUMC, Amsterdam, The Netherlands
  2. 2Jan van Breemen Research Institute/READE, Amsterdam, The Netherlands
  3. 3Manchester, UK
  4. 4Department of Rheumatology, VU University Medical Center, Amsterdam, The Netherlands
  5. 5BHF Glasgow Cardiovascular Research Centre, University of Glasgow, Glasgow, UK
  6. 6Institute of Infection, Immunity and Inflammation, University of Glasgow, Glasgow, UK
  1. Correspondence to Dr Michael T Nurmohamed, Department of Rheumatology and Internal Medicine, VUMC, PO Box 7057, Room 4A32, Amsterdam 1007 MB, The Netherlands; m.nurmohamed{at}reade.nl

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In their comments about our systematic review on cardiovascular (CV) comorbidities in patients with psoriatic arthritis,1 Gonzalez-Gay et al2 point out that in their studies, in contrast to our table 4, they did demonstrate a correlation between inflammatory parameters and flow-mediated dilatation …

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