Livedoid vasculopathy (LV) is a chronic recurrent painful cutaneous disease characterised by foci of purpuric lesions that convert to shallow ulcerations that progress to atrophic scar-like plaques mixed with telangiectasia and hyperpigmentation.1 2 Although of unknown aetiology, LV strongly correlates with immune complex associated diseases that lead to dermal vessel occlusion,3 and its histology consistently exhibits inflammatory infiltrates.4 Laboratory abnormalities consist essentially of a hypercoagulable state.5 Treatment of LV is usually empirical and based on immunosuppression with high-dose steroids, azathioprine, methotrexate and anticoagulation.6 In this report, we present a patient …