• anti-tumour necrosis factor α
• anorexia nervosa
• juvenile idiopathic arthritis

Tumour necrosis factor α (TNFα) is believed to have a pivotal role in the pathogenesis of many forms of inflammatory arthritis, and anti-TNFα therapies are now licensed and recommended for the treatment of refractory rheumatoid arthritis and juvenile idiopathic arthritis (JIA).^1,2 An important role for TNFα in the pathophysiology of anorexia nervosa has also been postulated, although authors differ on the precise mechanisms underlying its role.³ Some authors have suggested that TNFα may be mediating a stress response, whereas others have proposed that raised levels of TNFα in these patients may be secondary to anorexia rather than a cause.^4,5 Although the role of anti-TNFα therapies in the management of inflammatory arthritis is well recognised, we are not aware of experience with the use of anti-TNFα treatment in the management of anorexia nervosa. We describe the effect of such treatment prescribed to a woman with refractory JIA and anorexia nervosa.