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Tumour necrosis factor α (TNFα) is believed to have a pivotal role in the pathogenesis of many forms of inflammatory arthritis, and anti-TNFα therapies are now licensed and recommended for the treatment of refractory rheumatoid arthritis and juvenile idiopathic arthritis (JIA).1,2 An important role for TNFα in the pathophysiology of anorexia nervosa has also been postulated, although authors differ on the precise mechanisms underlying its role.3 Some authors have suggested that TNFα may be mediating a stress response, whereas others have proposed that raised levels of TNFα in these patients may be secondary to anorexia rather than a cause.4,5 Although the role of anti-TNFα therapies in the management of inflammatory arthritis is well recognised, we are not aware of experience with the use of anti-TNFα treatment in the management of anorexia nervosa. We describe the effect of such treatment prescribed to a woman with refractory JIA and anorexia nervosa.
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