EDITORIAL
Why is gout so poorly managed?
1 Professor of Medicine (Rheumatology), Hospital General Universitario de Alicante, Universidad Miguel Hernández, Alicante, Spain
2 Staff Rheumatologist, Hospital General Universitario de Alicante, Alicante, Spain
Correspondence to:
Professor Eliseo Pascual, Sección de Reumatología, Hospital General Universitario de Alicante, Maestro Alonso 109, 03010 Alicante, Spain; pascual_eli@gva.es
Accepted 9 July 2007
See linked article, p1311
Abbreviations: EULAR, European League Against Rheumatism; GP, general practitioner; MSU, monosodium urate; RA, rheumatoid arthritis; SUA, serum uric acid
Keywords: gout; monosodium urate crystals; quality of care
| The first 150 words of the full text of this article appear below. |
Gout has been recognised since ancient times, and we currently have a deep understanding of its pathophysiology. The disease results from a deposit of monosodium urate (MSU) crystals in joint structures and in other, generally periarticular, sites in the form of tophi. High serum uric acid (SUA) is required for the formation of these crystals. The most characteristic features of gout are acute attacks of joint inflammation, which frequently occur at the first metatarsophalangeal joint, although their occurrence in other joints and in bursae is also very common. Oligoarticular, polyarticular, and more protracted and lingering forms of the disease occur, and if untreated or poorly managed, the disease can become very persistent and disabling. Fortunately, we now have highly effective drugs enabling us to deal with gouty joint inflammation and to prevent their recurrence.1 An unequivocal diagnosis can be obtained by identifying MSU crystals in joint fluid obtained either during
Relevant Article
- Concordance of the management of chronic gout in a UK primary-care population with the EULAR gout recommendations
- Edward Roddy, Weiya Zhang, and Michael Doherty
Ann Rheum Dis 2007 66: 1311-1315.[Abstract] [Full Text] [PDF]
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