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Reactive or infectious arthritis
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Why reactive arthritis? |
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A role for bacterial infection in the aetiology of
inflammatory arthritis has been suspected for many years. Yet over that relatively long period of time only a few acute or chronic arthritides have been unequivocally linked to an infectious agent; these include septic arthritis, rheumatic fever, and, more recently, Lyme arthritis. The term reactive arthritis was first introduced to describe the association between Yersinia enterocolitica
infection and arthritis, and it was intended to differentiate this form
of acute, non-suppurative arthritis, which is characterised by negative
joint culture, from infectious, purulent arthritis; the differentiation
was meant to suggest an underlying sterile immune mediated
pathomechanism.1 A few years later, immediately after
discovery of the association between HLA-B27 and ankylosing spondylitis
and Reiter's syndrome, the term reactive arthritis was also related to
this genetic marker; at this time the term was more strictly applied to
the HLA-B27 associated reactive arthritides, following infections with
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Degenerative joint disease
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